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Selank — 10mg

Selank — 10mg

$64.99 Regular Price
$54.99Sale Price
Pack Size
Single Vial
10-Pack (Ships in 2 Weeks)
Quantity

Research-grade Selank, a synthetic heptapeptide (Thr-Lys-Pro-Arg-Pro-Gly-Pro) derived from the immunomodulatory peptide tuftsin. Studied for anxiolytic properties, GABAergic modulation, and neuroimmune signaling without sedative effects or dependence profiles. 10mg lyophilized powder, 99%+ purity verified by Janoshik Analytical via HPLC and mass spectrometry.


  • For laboratory research use only.

  • Not for human or veterinary use.

  • Not intended for diagnosis, treatment, cure, or prevention of any disease.

  • Use only in controlled laboratory settings by qualified personnel following appropriate safety procedures.

  • Selank is a synthetic heptapeptide developed at the Institute of Molecular Genetics of the Russian Academy of Sciences. It is an analog of tuftsin, a naturally occurring immunomodulatory tetrapeptide derived from the heavy chain of human immunoglobulin G (IgG). The C-terminal Pro-Gly-Pro extension enhances metabolic stability. Unlike benzodiazepines, Selank modulates GABA-A receptors allosterically without causing sedation, cognitive impairment, or dependence in preclinical models. It is frequently studied alongside Semax, another Russian-developed neuropeptide.


    BENEFITS


    • Anxiolytic research — studied for GABA-A receptor allosteric modulation without sedation

    • Immunomodulation — investigated for T helper cell cytokine balance and IL-6 expression modulation

    • Nootropic research — explored for BDNF expression enhancement and enkephalin metabolism

    • No dependence profile — unlike benzodiazepines, no habituation or withdrawal in preclinical models

    • Tuftsin-derived — retains immunomodulatory properties of the parent peptide with enhanced stability


    WHAT RESEARCHERS LOOK AT


    • GABA-A receptor allosteric modulation and inhibitory postsynaptic current enhancement

    • Monoamine neurotransmitter metabolism (serotonin) in stress models

    • IL-6 expression modulation and T helper cell cytokine balance

    • Enkephalin-degrading enzyme inhibition and endogenous opioid system effects

    • Comparative analysis vs. benzodiazepine anxiolytics on cognitive function preservation

Frequently Paired With

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